Hydrazine-induced liver toxicity is enhanced by glutathione depletion but is not mediated by oxidative stress in HepG2 cells.

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Title:	Hydrazine-induced liver toxicity is enhanced by glutathione depletion but is not mediated by oxidative stress in HepG2 cells.
Author(s):	Olthof, E.D.; Tostmann, A. ; Peters, W.H.M. ; Schaap-Roelofs, H.M.J. ; Wagener, F.A.D.T.G. ; Scharstuhl, A. ; Dekhuijzen, P.N.R. ; Boeree, M.J.
Publication year:	2009
Source:	International Journal of Antimicrobial Agents, vol. 34, iss. 4, (2009), pp. 385-386
ISSN:	0924-8579
DOI:	https://doi.org/10.1016/j.ijantimicag.2009.06.003
Publication type:	Article / Letter to editor
Please use this identifier to cite or link to this item : https://hdl.handle.net/2066/80431
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Subject:	IGMD 9: Renal disorder N4i 3: Poverty-related infectious diseases NCMLS 3: Tissue engineering and pathology NCMLS 5: Membrane transport and intracellular motility ONCOL 3: Translational research ONCOL 5: Aetiology, screening and detection
Organization:	Gastroenterology Pulmonary Diseases Internal Medicine Pharmacology-Toxicology Dentistry
Former Organization:	Pharmacology/Toxicology
Journal title:	International Journal of Antimicrobial Agents
Volume:	vol. 34
Issue:	iss. 4
Page start:	p. 385
Page end:	p. 386

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