Publication year
2019Source
Hormone and Metabolic Research, 51, 7, (2019), pp. 414-418ISSN
Publication type
Article / Letter to editor

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Organization
Internal Medicine
Journal title
Hormone and Metabolic Research
Volume
vol. 51
Issue
iss. 7
Page start
p. 414
Page end
p. 418
Subject
Radboudumc 16: Vascular damage RIMLS: Radboud Institute for Molecular Life SciencesAbstract
The mitochondrial enzyme succinate dehydrogenase (SDH) acts as a tumor suppressor. Biallelic inactivation of one of the genes encoding for SDH subunits (collectively named SDHx) leads to complete loss of the protein function and the development of diverse group of tumors. Pheochromocytomas-paragangliomas are the prime example of hereditary tumors caused by SDH deficiency. In this review, we discuss the roles of imaging examinations, and illustrate new insights into genotype-imaging phenotype relationships.
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- Academic publications [227864]
- Faculty of Medical Sciences [86218]
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