EZH1/2 function mostly within canonical PRC2 and exhibit proliferation-dependent redundancy that shapes mutational signatures in cancer
Publication year
2019Source
Proceedings of the National Academy of Sciences USA, 116, 13, (2019), pp. 6075-6080ISSN
Publication type
Article / Letter to editor

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Organization
Proteomics and Chromatin Biology
Molecular Biology
Journal title
Proceedings of the National Academy of Sciences USA
Volume
vol. 116
Issue
iss. 13
Page start
p. 6075
Page end
p. 6080
Subject
Molecular Biology; Proteomics and Chromatin BiologyThis item appears in the following Collection(s)
- Academic publications [234237]
- Electronic publications [117187]
- Faculty of Science [34566]
- Open Access publications [84231]
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