RAGE does not contribute to renal injury and damage upon ischemia/reperfusion-induced injury.
Publication year
2012Source
Journal of Innate Immunity, 4, 1, (2012), pp. 80-85ISSN
Publication type
Article / Letter to editor

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Organization
Physiology
Pathology
Journal title
Journal of Innate Immunity
Volume
vol. 4
Issue
iss. 1
Page start
p. 80
Page end
p. 85
Subject
NCMLS 5: Membrane transport and intracellular motility IGMD 9: Renal disorder; ONCOL 3: Translational researchAbstract
The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury.
This item appears in the following Collection(s)
- Academic publications [202914]
- Electronic publications [101091]
- Faculty of Medical Sciences [80065]
- Open Access publications [69755]
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