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Browsing by Author "Smeets, B."
Now showing items 1-36 of 36
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Inhibition of mTOR delayed but could not prevent experimental collapsing focal segmental glomerulosclerosis ; Eymael, J.; Sharma, Shagun; ; Willemsen, B.K.T.; ; ; ; ; Jansen, J.; et al.2020, Article / Letter to editor (Scientific Reports, vol. 10, (2020), article 8580)
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A Comprehensive Analysis of Ontogeny of Renal Drug Transporters: mRNA Analyses, Quantitative Proteomics, and Localization Cheung, K.W.K.; Groen, B.D.; Spaans, E.; Borselen, M.D. van; Bruijn, A. de; Simons-Oosterhuis, Y.; Tibboel, D.; Samsom, J.N.; Verdijk, R.M.; ; et al.2019, Article / Letter to editor (Clinical Pharmacology and Therapeutics, vol. 106, iss. 5, (2019), pp. 1083-1092)
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CD44 is required for the pathogenesis of experimental crescentic glomerulonephritis and collapsing focal segmental glomerulosclerosis Eymael, J.; Sharma, Shagun; ; ; ; Florquin, S.; ; Willemsen, B.K.T.; Sharma, V.; ; et al.2018, Article / Letter to editor (Kidney International, vol. 93, iss. 3, (2018), pp. 626-642)
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Detection of activated parietal epithelial cells on the glomerular tuft distinguishes early focal segmental glomerulosclerosis from minimal change disease ; Stucker, F.; ; Brocheriou, I.; Ronco, P.; Grone, H.J.; D'Agati, V.; Fogo, A.B.; ; Fischer, H.P.; et al.2014, Article / Letter to editor (American Journal of Pathology, vol. 184, iss. 12, (2014), pp. 3239-3248)
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Albumin is recycled from the primary urine by tubular transcytosis Tenten, V.; Menzel, S.; Kunter, U.; Sicking, E.M.; Roeyen, C.R. van; Sanden, S.K.; Kaldenbach, M.; Boor, P.; Fuss, A.; Uhlig, S.; et al.2013, Article / Letter to editor (Journal of the American Society of Nephrology, vol. 24, iss. 12, (2013), pp. 1966-1980)
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Proximal tubular cells contain a phenotypically distinct, scattered cell population involved in tubular regeneration ; Boor, P.; ; Sharma, S.V.; Jirak, P.; ; Berger, K.; Bornemann, J.; Gelman, I.H.; Floege, J.; et al.2013, Article / Letter to editor (Journal of Pathology, vol. 229, iss. 5, (2013), pp. 645-659)
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Subtotal ablation of parietal epithelial cells induces crescent formation. Sicking, E.M.; Fuss, A.; Uhlig, S.; Jirak, P.; Dijkman, H.; Wetzels, J.; Engel, D.R.; Urzynicok, T.; Heidenreich, S.; Kriz, W.; et al.2012, Article / Letter to editor (Journal of the American Society of Nephrology, vol. 23, iss. 4, (2012), pp. 629-640)
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Primary cultures of glomerular parietal epithelial cells or podocytes with proven origin. Kabgani, N.; Grigoleit, T.; Schulte, K.; Sechi, A.; Sauer-Lehnen, S.; Tag, C.; Boor, P.; Kuppe, C.; Warsow, G.; Schordan, S.; et al.2012, Article / Letter to editor (PLoS One, vol. 7, iss. 4, (2012), article e34907)
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Parietal epithelial cells participate in the formation of sclerotic lesions in focal segmental glomerulosclerosis ; Kuppe, C.; Sicking, E.M.; Fuss, A.; Jirak, P.; ; Endlich, K.; ; Grone, H.J.; Floege, J.; et al.2011, Article / Letter to editor (Journal of the American Society of Nephrology, vol. 22, iss. 7, (2011), pp. 1262-1274)
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Renal progenitor cells contribute to hyperplastic lesions of podocytopathies and crescentic glomerulonephritis. ; Angelotti, M.L.; Rizzo, P.; Dijkman, H.; Lazzeri, E.; ; Ballerini, L.; Parente, E.; Sagrinati, C.; Mazzinghi, B.; et al.2009, Article / Letter to editor (Journal of the American Society of Nephrology, vol. 20, iss. 12, (2009), pp. 2593-2603)
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